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However, weeks after the onset of ischemia, the chronic phase leads to additional tissue damage and may be the result of delayed neurodegeneration triggered by oxidative stress and immune activation

1 In silico analysis of the impact of R152H mutation on GPX4 (GPX4 U46C PDB: 2OBI) structure

The global methylation status showed significantly increased 5-mC levels in GCLC knockdown cells (Fig

Figure 1 The first process consists in the de novo synthesis of GSH from its component amino acids, through two ATP-consuming enzymatic reactions

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