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Physiologically occurring cell death, referred to as programmed cell death (PCD), is essential for appropriate embryonic development of multicellular organisms as well as for tissue remodeling during adulthood [1]

Importantly, therapeutic strategies should target not only the source of initiation and/or progression of the disease but also self-amplifying mechanisms that sustain emphysema themselves, namely airspace enlargement, ECM breakdown, and progressive mechanical failure, all of which should hinder the self-perpetuation of tissue damage and stabilize lung architecture

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In some cases, the dementia and other neurologic symptoms caused by vitamin B 12 deficiency can be reversed by vitamin treatment (42), but reversibility seems to be dependent upon the duration of the associated neurologic complications (41)