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Resolvins, lipoxins, maresins, and protectins are specialized pro-resolving mediators (SPM), which are downstream derivatives of PUFA (133)

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TMAO-induced inflammation and endothelial dysfunction in human umbilical vein endothelial cells (HUVECs) can be reversed by treatment with the ROS inhibitor N-acetylcysteine (NAC) or by siRNA-mediated knockdown of NLRP3, suggesting that TMAO exerts its pro-inflammatory effects via the ROS/NLRP3 inflammasome [146]

Cold solutions can cause the peptide to precipitate improperly