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In a recent article, our research group reported that Nogo-A could also interact with astrocytes, giving them a more antisynaptogenic profile, impairing their mechanisms of synaptic regulation, such as glutamate uptake ( Figure 2 )

The lack of statistically significant effect on this outcome, which was repeatedly demonstrated to improve in the CVOTs with GLP-1 RAs [12], can be explained by the irregularity of UACR sampling in real-world setting compared with CVOTs