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This is the axis that links the remaining mechanisms: protection of cells against damage while simultaneously supporting vessel formation and the migration of repair cells
This explains why high levels of oxidative stress is implicated not just in neurodegenerative conditions but cardiovascular diseases as well
It can also activate transient receptor potential vanilloid 1 (TRPV1) channels, altering visceral pain thresholds, and participate in the regulation of the hypothalamic-pituitary-adrenal (HPA) axis and stress responses
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