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This FAK-paxillin signaling axis connects BPC-157's molecular pharmacology to its observed tissue-level effects in a mechanistically coherent way: a compound that enhances endothelial FAK activation will simultaneously promote cell survival (resistance to anoikis), cell migration (capillary sprouting), and cell-matrix adhesion (structural integrity of new vessels)

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10.1080/15548627.2017.1291113 Autophagy 273 YamashitaS

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Given its origins from a gastric protein, gastroenterology is another primary area of investigation