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Chronic low-grade inflammation, increasingly recognized as a contributor to numerous age-related health conditions, may be partially mediated by inadequate antioxidant defenses including suboptimal glutathione status

It is studied for its anti-inflammatory effects via the melanocortin receptor system and through downstream inhibition of NF-B

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It also regulates matrix metalloproteinases (MMPs) and their inhibitors (TIMPs), ensuring controlled tissue remodeling and balanced extracellular matrix turnover[1]