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Endothelial dysfunction is a significant component of HFpEF pathophysiology, and glucagon-like peptide-1 receptor (GLP-1R) agonists have shown potential benefits in improving endothelial function

In line with this, we demonstrate that autophagy is essential for the up-regulation of an innate immune pathway in glial cells after axon injury, characterized by the induction of virus-induced RNA 1 ( vir-1 )

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Exposures At least 2 prescriptions of a GLP-1 RA given 6 months apart