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Supporting this assumption, it was demonstrated in mice that SFN dampened the clinical severity of experimental arthritis by inhibiting the production of cartilage destructive metalloproteinases and the expression of IL-17 and TNF- ( Also, of high clinical importance may be our second finding, namely that SFN has differential effects on untransformed human T-cells (decrease of GSH and immunosuppression) and Jurkat T-leukemia cellsprototypical immature transformed T-cells (increase of GSH and cytotoxicity)

Fifth, serum levels of trimethylamine N-oxide (TMAO), a key metabolite of L-carnitine associated with cardiovascular disease risk, were not evaluated

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Neurosci Lett (2009) 465(1):615.10.1016/j.neulet.2009.08.081 58 HartungJEEskewOWongTTchivilevaIEOladosuFAOBuckleySCet alNuclear factor-kappa B regulates pain and COMT expression in a rodent model of inflammation