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reported an increase in the levels of dysfunctional mitochondria and mitochondrial membrane proteins, translocase of the outer mitochondrial membrane 20 (Tom20), translocase of the inner mitochondrial membrane 23 (Tim23), and porin in the ASD temporal cortex without a change in the transcription of genes, such as PARK2 , responsible for the activation of mitophagy

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Even if BPC-157 survives gastric acid, pepsin and pancreatic proteases remain active in the duodenum and small intestine at physiological pH and will hydrolyze unprotected peptide bonds