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I think there's still a lot of confusion around how and why the GLP1 medicines work

JBSNF-000088, a small-molecule analog of NA, is able to be bound to NNMT and to be methylated in the NNMT structure, with SAM being correspondingly demethylated to SAH [58]

The starting point coincided with the approval date of the first GLP-1 RA, exenatide (BYETTA), April 28, 2005

Not quite as good as if you had bought Eli Lilly at the same time (+106%), but much better than the other major GLP-1 stock, Novo Nordisk (incredibly, down 4% since the beginning of 2023)

The overproduction of ROS leads to different kinds of downstream impairments, including ferroptosis