The reasonable reading is that cagrilintides gastrointestinal effects are mechanism-based and generally manageable with titration in the studied populations, but that manageable in trials is not the same as fully characterized for long-term real-world use, and the delayed-emptying mechanism itself carries context-specific cautions that should not be waved away
and A.T.-V
The authors speculate that the reduced activity of the redox-sensitive protein tyrosine phosphatase (PTP)1B and the higher concentration of irreversible oxidized PTP1B could be, at least in part, responsible for this effect [12]
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