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Z., Kaur, H., Halliwell, B., Li, X

To contextualize the complexity of these interactions, we present an integrated mechanistic framework (Fig

The result is a peptide that retains and enhances the cognitive/neuroprotective properties of the ACTH fragment without triggering cortisol release

Most studies have focused on HO-1 affecting diabetic complications through its antioxidant, anti-apoptotic, and anti-inflammatory effects, but HO-1 has also shown a role in programmed cell death that cannot be ignored