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The anti-inflammatory potential of BL has also been validated in neurodegenerative disease models: in experimental autoimmune encephalomyelitis, BRNPs attenuate antigen-presenting cell maturation by scavenging ROS generated during dendritic cell and macrophage antigen uptake, thereby inhibiting nave CD4 + T cell differentiation into Th17 cells and reducing pro-inflammatory Th17 populations (Kim et al., 2021)

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