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Surgical trauma can transiently elevate total cfDNA, necessitating careful timing and serial sampling

Genetic polymorphisms may be a contributing factor as to why its hard to precisely pin down a healthy MMA threshold

In addition, the inhibition of aspartate aminotransaminase GOT1 promotes labile iron release in PDAC through NCOA4-dependent ferritinophagy, highlighting GOT1 as a therapeutic target through the promotion of PDAC ferroptosis [24]

They also illustrate an important distinction: increasing a laboratory measure of glutathione is not automatically proof that supplementation prevents disease, improves longevity or produces a noticeable clinical benefit