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It has been proposed that accumulation of these populations lead to more propionate production that is absorbed into the liver and hence progression to MASLD

The presence of NO in the follicular fluid and the expression of NO synthase in follicles and corpus luteum have been reported [19, 141, 143, 145]

320 These observations were confirmed in ob/ob mice and high-fat-fed mice when they were treated with a GIPR antagonist, and also in mice lacking GIP-secreting K cells, confirming the important role of GIP in fat accumulation