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1 Introduction Sepsis-associated encephalopathy (SAE) represents a severe complication of sepsis, defined by diffuse brain dysfunction without direct central nervous system (CNS) infection ( Oxidative stress is central to SAE pathogenesis, driven by an imbalance between excessive reactive oxygen species (ROS) production and impaired antioxidant defense systems ( The antioxidant defense system, especially GSH metabolism, is linked to neurodegenerative diseases ( Proteomics and metabolomics enable systematic profiling of protein expression and metabolic signatures

A.et al (2023b)

For cases involving possible nerve issues, we may refer for nerve studies, like electromyography (EMG) or nerve conduction tests

Systemic hyperinflammation, hypercytokinemia, and the development of cytokine storms may affect intestinal microbiota (Jung and Jung 2022)