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Conclusions Our study reveals the critical role of hepsin, a type II transmembrane serine protease, in protecting against APAP-induced liver injury by decreasing cellular GJ abundance in response to reactive oxygen stress in the liver

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The indirect inhibition of GSK3 activity occurs through the activation of the AKT kinase family, resulting in inhibitory phosphorylation at the N-terminal region and suppression of phosphatases responsible for GSK3 dephosphorylation [38]