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However, this phosphorylation was blocked by the GLP-1-R-Antagonist Exendin(9-36), emphasizing the role of GLP-1-mediated signaling [16]

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While Glu9 of GLP-1 formed strong and persistent hydrogen bond and van der Waals interactions with R190 2.60 , Y152 1.47 , and to a lesser extent, Y148 1.43 , the polar side chain of Gln3 in oxyntomodulin only formed weak and very transient hydrogen bonds with these residues, and van der Waals interactions only with Y148 1.43 (Movie S1, Supplementary Table 3)

These findings highlight bile acids as dynamic neuromodulators originating from the gut microbiota that are capable of both protective and detrimental effects on neuronal function