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Thus, there is an urgent need to characterize the specific neuropathological mechanisms underlying the impacts of adolescent THC exposure on neuropsychiatric risk and to identify potential intervention strategies aimed at preventing or reversing these outcomes [10, 11]

Author contributions XG was the chief investigator in charge of this trial program

Maintaining consistent treatment is crucial during this time, as the cumulative hormonal adjustments support normal follicular development

Extrapolating from semaglutide and tirzepatide data is reasonable given shared molecular characteristics (large protein-based structures, poor oral bioavailability), but it is not the same as having direct evidence

Abbreviations: ce, cytoplasmatic extensions