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The flux of cysteine and the end products are driven by how much methionine is eaten, as well as how much glutathione is needed to combat oxidative stress in cells.[ref][ref] Homocysteine thiolactone: Damaging endothelial cells, increasing inflammation Homocysteine can also become homocysteine thiolactone when a thiol group is attached

The resulting gene lists were annotated and filtered for significantly differentially up- and down-regulated genes

Alterations in the HLA-B*57:01 immunopeptidome by flucloxacillin and immunogenicity of drug-haptenated peptides