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First, TAZ interacts with RORt to increase its transcriptional activity while competitively relieving Foxp3-mediated repression, thereby coordinately upregulating Th17-associated gene expression and promoting Th17 cell development

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The combined treatment reduced HbA1c to a greater degree considering it decreased by 2.2% with the combination of semaglutide and cagrilinide, 1.8% with semaglutide alone, and 0.9% with cagrilintide [2]

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