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The peptide reduces myofibroblast differentiation by downregulating -smooth muscle actin (-SMA) and collagen type I alpha 1 (Col1a1) expression, key markers of activated fibroblasts

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[17,41] Scientific evidence (efficacy): Unlike BPC-157, the original TB4 molecule has also undergone clinical studies in humans, which suggest its ability to suppress inflammation and prevent cell death (apoptosis)

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