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glp-1 mitochondria improves the neuronal supportive ability of astrocytes in Alzheimer's disease by regulating mitochondrial dysfunction via the cAMP/PKA pathway Schematic on the nutrient-induced stimulation
glp-1 mitochondria improves the neuronal supportive ability of astrocytes in Alzheimer's disease by regulating mitochondrial dysfunction via the cAMP/PKA pathway Schematic on the nutrient-induced stimulation
glp-1 mitochondria improves the neuronal supportive ability of astrocytes in Alzheimer's disease by regulating mitochondrial dysfunction via the cAMP/PKA pathway Schematic on the nutrient-induced stimulation
glp-1 mitochondria improves the neuronal supportive ability of astrocytes in Alzheimer's disease by regulating mitochondrial dysfunction via the cAMP/PKA pathway Schematic on the nutrient-induced stimulation
glp-1 mitochondria

glp-1 mitochondria improves the neuronal supportive ability of astrocytes in Alzheimer's disease by regulating mitochondrial dysfunction via the cAMP/PKA pathway Schematic on the nutrient-induced stimulation

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Description

Martensson J, Jain A, and Meister A (1990)

glp-1 mitochondria improves the neuronal supportive ability of astrocytes in Alzheimer's disease by regulating mitochondrial dysfunction via the cAMP/PKA pathway Schematic on the nutrient-induced stimulation

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glp-1 mitochondria improves the neuronal supportive ability of astrocytes in Alzheimer's disease by regulating mitochondrial dysfunction via the cAMP/PKA pathway Schematic on the nutrient-induced stimulation

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glp-1 mitochondria improves the neuronal supportive ability of astrocytes in Alzheimer's disease by regulating mitochondrial dysfunction via the cAMP/PKA pathway Schematic on the nutrient-induced stimulation

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glp-1 mitochondria improves the neuronal supportive ability of astrocytes in Alzheimer's disease by regulating mitochondrial dysfunction via the cAMP/PKA pathway Schematic on the nutrient-induced stimulation

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glp-1 mitochondria improves the neuronal supportive ability of astrocytes in Alzheimer's disease by regulating mitochondrial dysfunction via the cAMP/PKA pathway Schematic on the nutrient-induced stimulation
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