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(C) IH growth to adjacent cells was significantly reduced (Students t-test p0.05) in gtr1 strains compared to WT at 48 hpi

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We would like to express their sincere gratitude to the relevant funding committees for the financial support

Aconitase inactivation and oxidative stress have been noted in other neuropsychiatric and neurodegenerative disorders with known mitochondrial involvement, including schizophrenia, 50, 51 Huntington's disease 52 and Parkinson's disease

Our findings indicated that targeting ACSL4 or, alternatively, upregulating GPX4 or FSP1 may help mitigate AKI by reducing ferroptosis, offering potential therapeutic strategies for oxalate-induced AKI