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R., and Spector, S

Methods This propensity-score matched cohort study analyzed TriNetX US network data from November 2023 through April 2025

They stimulate glucose-dependent insulin release from the pancreas, slow gastric emptying (prolonging a feeling of satiety), inhibit post-meal glucagon release (glucagon stimulates liver and muscle cells to convert stored glycogen back into glucose, raising blood glucose levels) and reduce the desire for food intake

Mechanism of Action GLP-1R agonism enhances glucose-dependent insulin secretion, suppresses glucagon, delays gastric emptying, and reduces appetite GIPR agonism amplifies insulin secretion additively

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