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Combined triad-GSH metabolism model simulations: impact of quinone metabolism on glutathione homeostasis The model was used to investigate how single vs constant 50 M exposure of doxorubicin influenced blood and cytosolic GSH and cysteine (Cys) levels, over a 10-hour time-span, shown in Fig

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Mitochondrial diseases are characterized by both a diminished capacity to produce ATP as well as an elevated production of reactive oxygen species (ROS) and reactive nitrogen species (RNS) [3]