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Antilipogenic action of synthetic C-terminal sequence 177-191 of human growth hormone

Results showed that the ingredient significantly increased NAD+ levels in senescent fibroblasts, activated the SIRT1 signaling pathway, reduced the expression of senescence-associated -galactosidase (SA--gal), decreased intracellular reactive oxygen species accumulation, and promoted collagen synthesis

doi: 10.3390/nu11020482 51

GPX4-IN-3 (26a) exhibits outstanding GPX4 inhibitory activity with a percent inhibition up to 71.7% at 1.0 M compared to 45.9% of RSL-3 [1]

It works by boosting nitric oxide production, improving circulation, and reducing oxidative stress