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First, H3K18la is frequently associated with transcriptional activation of pro-tumor programs and adverse phenotypes, as initially shown in the discovery study of histone lactylation and subsequently reported in multiple cancer contexts [83, 178, 310]

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Under physiological conditions, sulfite (S = +4) is endogenously produced in the mitochondria and then further oxidized to inorganic sulfate (SO 4 2 ) (S = +6), by molybdenum-dependent sulfite oxidase (SOUX), which resides in the intermembrane space of the mitochondria [54]

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