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Taken together, these results indicate that p53 can mediate the SLC7 A11/GPX4 signaling pathway to regulate trophoblast ferroptosis, which may lead to placental tissue angiogenesis disorders and participate in the pathogenesis of PE

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Clinical Diabetes: A Publication of the American Diabetes Association, 41 (3), 467-473, Bendotti, G., Montefusco, L., Lunati, M

The microbiome-associated alterations summarized in Table 1 suggest a potential mechanistic framework in which gut dysbiosis may contribute to peripheral immune dysregulation and influence central neuroinflammatory processes

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