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Discussion Beyond mRNA and histone methylation, there may be other MTases directly or indirectly inhibited by excess of SAM, adenine, and MTA

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Targeting Downstream Effectors of ER Activation While ER-targeted therapies are likely to suffer from some of the same risks as estrogen supplementation (115), targeting selected downstream effectors of ER may represent an opportunity to capture the metabolic benefits while bypassing adverse effects associated with estrogens