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However, increasing non-ionic surfactants and cholesterol beyond optimal levels (above 30 mg) led to reduced entrapment efficiency and larger particle sizes in niosomes, likely due to disrupted vesicular structures and potential formation of mixed micelles 56,57

In the animal model, BPC-157 inhibited the muscularization of pulmonary arteries, the pathological process that drives pulmonary hypertension

Phenazine virulence factor binding to extracellular DNA is important for Pseudomonas aeruginosa biofilm formation

This element has known effects on diabetes and cardiovascular disorders through its impact on inflammatory reactions (140)