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Besides, after an induced traumatic brain injury in mice, BPC 157 regimens (corresponding to those used in the present study) demonstrated a marked attenuation of damage with an improved early outcome and a minimal postponed mortality throughout a 24 h post-injury period

Key outcome measures included the latency to find the platform during training and the time spent in the target quadrant during probe trials, which are indicators of spatial memory retention [2]

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How does AOD 9604 work (mechanism of action) While full mechanistic clarity is still under investigation, here are the main proposed/examined pathways: It stimulates lipolysis (the breakdown of stored triglycerides into glycerol + free fatty acids) in fat cells (adipocytes)

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