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ALOXs are nonheme ferric enzymes that catalyze the oxidation of PUFAs to generate a series of secondary metabolites that in turn promote ferroptosis [27]

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In children with ASD, studies have demonstrated that glutathione metabolism can be improved with subcutaneously injected methylcobalamin and oral folinic acid ( N -acetyl-l-cysteine, a supplement that provides a precursor to glutathione, was effective in improving symptoms and behaviors associated with ASD ( Small ( N -acetyl-l-cysteine ( Several other treatments that have antioxidant properties ( These studies suggest that treatments that address oxidative stress may improve core and associated symptoms of ASD

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