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However, in the long term, the stressed heart undergoes maladaptive remodeling resulting in the development of fibrosis, LV dilatation, and dysfunction, thus predisposing individuals to heart failure (Tham et al, 2015

Moreover, we summarized the pharmacological progress of targeting ferroptosis in DKD in the hope of providing new therapeutic strategies for patients with DKD

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504,505 Defective cholesterol metabolism, such as in LXR-deficient T cells, impairs their proliferative capacity following activation

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