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Therefore, if cell lysis due to pore formation by Hld Se was driving GLP-1 release we would have observed toxicity in our studies

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In one study, intermittent fasting (i.e., alternate-day fasting) alleviated the increase of LPL expression in the brain of a mouse model of AD possibly by mediation of an increase in ketone body levels (i.e., -hydroxybutyrate) subsequent to the induced ketosis [130]

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