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[85] demonstrated that carnosine could abolish 4-HNE production and attenuate cartilage degeneration in the dog model of OA

Similarly, activating the Nrf2/HO-1 axis to enhance cellular antioxidant gene expression can hamper ferroptosis, although the potentially excessive iron release from high HO-1 expression must be considered (128)

At the same time, in older adults, the impact may extend to multiple neurotransmitter systems, including dopaminergic and glutamatergic pathways, leading to more complex neuropsychiatric manifestations [117]