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Plasticity of Excitatory Inputs to CA1 Interneurons in the Hippocampus Diverse Forms of Calcium-Dependent Plasticity in CA1 Interneurons The first intracellular study of plasticity of excitatory transmission to inhibitory neurons in the hippocampus aimed to reveal whether changes in excitability of interneurons could contribute to regular tetanus-induced LTP of field potentials (Taube and Schwartzkroin, Induction of plasticity in fast-spiking (FS) neurons from CA1 str
To investigate this, we introduced a mutation where cys115 was replaced with alanine, resulting in the inhibition of H 2 S-induced sulfhydration of Syvn1 in HL-1 cardiomyocytes (Fig
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