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The percentage of subjects with one deficit increased from 18 subjects (69.2%) to 19 subjects (73.1%) after 10 weeks, while the percentage of subjects with two deficits decreased from ten subjects (30.8%) to four subjects (15.4%) after 10 weeks intervention

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The trial results noted minor improvement exhibited in the subjects cholesterol profiles and glucose tolerance levels

The MyD88-dependent pathway activates NF-B and mitogen-activated protein (MAP) kinases, inducing the expression of inflammatory cytokine genes, while the TRIF-dependent pathway activates NF-B, IRF 3 and MAPKs inducing type I interferons and inflammatory cytokines