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This adaptive transcriptional program promotes angiogenesis, shifts the metabolic program towards glycolysis and maintains ECM homeostasis (116, 117)

Furthermore, one of the mechanisms by which L-Carnitine supplementation may reduce oxidative stress is through the preservation of mitochondrial function by enhancing catalase and superoxide, which are antioxidant enzymes (Le Borgne et al

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30 , representing genes negatively regulated in the fibroblast core serum response, was also strongly enriched among the genes upregulated by GSTO1 knockdown (Fig