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Additionally, TGF-1 promotes fibrosis by enhancing autophagic flux through increasing cellular autophagosomes, thus reducing the level of autophagy in fibrotic tissues, which may be a potential target for anti-fibrotic therapy (Weiskirchen and Tacke, 2019

Radical phosphate transfer mechanism for the thiamin diphosphate- and FAD-dependent pyruvate oxidase from Lactobacillus plantarum

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Glaucomatous outflow pathway and oxidative stress

The in vivo antibacterial activity of the lysozymes is limited by numerous general cell-wall modifications hindering the enzyme to reach its substrate [44]