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Genome-wide allelotyping and CGH have directly confirmed the presence of monoclonal parathyroid neoplasms in uremic patients with refractory secondary hyperparathyroidism whereas the candidate gene approach has led to only modest results

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The lipid peroxide thus formed stimulates the synthesis of cyclooxygenase, prostaglandin and thromboxane which in turn causes increased platelets aggregation leading to vascular complications [20]

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