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Furthermore, propofols neuroprotective role in the stroke model involves the inhibition of the NLRP1-caspase-1-caspase-6 inflammatory pathway [77]

GLP-1R expression on intraepithelial lymphocytes and gut macrophages likely mediates these local effects, supporting the therapeutic rationale for GLP-1R-targeted nanotherapies in reducing steroid dependence [400, 401]

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