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Despite this, the salient role of overall vagal signaling in energy homeostasis has been established, as surgical or chemical ablation of vagal afferent signaling decreases acute sensitivity to intestinal nutrients, leading to increased food intake and reduced NTS neural activation 23,24,42,43,44
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Specifically, it up-regulates CPT1 mRNA expression, thereby promoting mitochondrial fatty acid transport and enhancing -oxidation activity (25, 26)
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