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We exposed cell line AC01 cells derived from mouse aortic smooth muscle to 0.1 M nicotine [32], and performed exhaustive gene expression analysis using DNA microarray for gene expression after 48 h

Still, a strength of this study is the inclusion of any measure of secondhand smoke exposure, which was not available for inclusion in the NHIS study and has not been examined as a potential confounder of the relationship between childhood smoking and COPD risk in prior work

Rolling tobacco brands also incurred increases ranging from 70p to 1.15

[5] The anti-tobacco movement did not have much effect in the early years of the Nazi rule

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