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The stimulation of smoke-exposed epithelial cells with PAMPs, such as lipopolysaccharide (LPS) or dsRNA as well as exposure to human rhinovirus, nontypeable Haemophilus influenzae (NTHI), Staphylococcus aureus and Moraxella catarrhalis attenuated the in vitro production of potent pro-inflammatory mediators, in particular pathogen-induced neutrophil-mobilizing cytokines [136,153,154,155,156]

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Platelet activity and aggregation were enhanced, as well as increased levels of platelet adhesion markers CD41, CD42b, and CD62p were measured after e-vapor exposure [111]

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