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CONCLUSIONS Our findings support a model in which cigarette smoke exposure is associated with MKK3/p38 MAPK activation and PRKN Ser131 phosphorylation, accompanied by impaired PRKN-dependent mitophagy and increased epithelial senescence markers in vitro and in vivo

CS-induced MFN/OPA1 expression causes hyperfusion of mitochondria accompanied by impaired stress resistance and cellular senescence in lung epithelial cells [22]

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