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Smoking is the major risk factor for COPD, and cigarette smoke (CS)-induced oxidative stress can lead to the damage of airway epithelium and compromise the barrier function, which is the initial step and pathological basis of COPD progression [3,4,5,6]

The concentration of hemoglobin-bound CO (CO-Hb) is intermittently higher in the blood of smokers than nonsmokers, and at levels known to reduce oxidative stress and inflammation

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It has outstanding fuel economy