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Conclusion Smoking-related erythrocytosis appears to be EPO-independent or EPO-suppressed, suggesting alternative mechanisms such as altered erythroid sensitivity, circadian EPO dynamics, or direct effects on erythropoiesis White blood cells: a patterned inflammatory signal If the red cell compartment reflects hypoxia, the white cell compartment reflects inflammation

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Discussion The bioinformatics methods used in the analyses of genomic sequences and gene expression profiles of genes and regulatory elements can identify the changes that arise because of exposures to tobacco smoke